D
dmanc227 said:In every other thread i've looked at containing Intelligent Design the debate has turned into the argument of whether or not Intelligent Design is correct or plausible. I'm more interested in seeing (fact-based) arguments on why the teaching of intellegent design would help or harm students.
So basically, "should the public high school science curriculum include the study of the Theory of Intelligent Design"?
to start it off:
The Theory of Intelligent Design is religion based and would therefore impose one's religions over students who might not share the same opinions.
CaliNORML said:I think that presenting the observable evidence of human intelligence and asking the question why human beings are more intelligent than other species is a legitimate question and an important one.
It should stop at the question only, the reason humans have higher intelligence should be left open, not filled in with one side or the others speculation.
But not in this tread, which deals with whether or not ID should be taught in school.CaliNORML said:I think that presenting the observable evidence of human intelligence and asking the question why human beings are more intelligent than other species is a legitimate question and an important one.
CaliNORML said:Yes, but why did only humans develop this brain and how it functions setting us apart from animals?
KMS
Vandeervecken said:There is no such thing as, "The Theory of Intelligent Design." ID does not meet the criteria of a scientific theory. It is merely one of millions of hypotheses out there.
Teaching it would establish religion. It most certainly is nothing but religion.
If "much debate" is equivalent to "pretty much no debate at all", you'd be right. The only debate about this is going on outside the scientific community, in the realm of those who are ignorant about Evolutionary Theory. Notice that you take that link from a site whose stated mission is to "integrate science and faith". They have a clear agenda, and they are in fact wrong in this article.HTColeman said:Well, there is much debate over whether evolution qualifies as a theory.
http://www.grisda.org/origins/04004.htm
Untrue. Evolutionary Theory is predictive, but not in the manner that you are thinking. Not only that, predictive capabilities are not inherent to the definition of a theory.It cannot be tested and it does not have "predictive qualities". Scientists cannot begin to say that under 'x' conditions 'y' will evolve into 'z' over 't' amount of years. Also it has circular reasoning;
Wikipedia said:In various sciences, a theory is a logically self-consistent model or framework for describing the behavior of a certain natural or social phenomenon, thus either originating from or supported by experimental evidence (see scientific method). In this sense, a theory is a systematic and formalized expression of all previous observations made that is predictive, logical, testable, and has never been falsified.
"The concept of natural selection by survival of the fittest is the basic evolutionary mechanism. This concept does not qualify as a scientific principle, since fitness is equivalent to survival. Here we have a case of circular reasoning; no consistency or predictive value can be tested. According to this idea, organisms have survived through the evolutionary process because they are better fit, and the way one tells they are better fit is that they survive."
Engimo said:If "much debate" is equivalent to "pretty much no debate at all", you'd be right. The only debate about this is going on outside the scientific community, in the realm of those who are ignorant about Evolutionary Theory. Notice that you take that link from a site whose stated mission is to "integrate science and faith". They have a clear agenda, and they are in fact wrong in this article.
Untrue. Evolutionary Theory is predictive, but not in the manner that you are thinking. Not only that, predictive capabilities are not inherent to the definition of a theory.
Wikipedia by Engimo said:In various sciences, a theory is a logically self-consistent model or framework for describing the behavior of a certain natural or social phenomenon, thus either originating from or supported by experimental evidence (see scientific method). In this sense, a theory is a systematic and formalized expression of all previous observations made that is predictive, logical, testable, and has never been falsified.
The "testability" that is referred to in science is not necessarily the ability to do an experiment that shows the accuracy of Evolution. In this case, testability is simply the ability of producing evidence that corroborates the theory - the fossil record following Evolutionary paths, etc.
Untrue. You can look at the conditions of the time and see why their adaptations made them better fit for survival. If there is a sudden flood in an area, those organisms that can breathe underwater or are fit to survive in a flooded environment will thrive. There is no circular reasoning, the animals survive because they were more fit, and they were more fit because of specific adaptations they had that correspond with the environmental conditions of the time.
HTColeman said:I take it that you are so involved in the scientific community that you are aware of the various debates within? Besides I simply said much debate, I didn't say where, and there is much debate.
So, how is it predictive? Furthermore, according to the definition from Wikipedia you posted, it is inherent to the definition of a theory.
That is not a test, that is what they believe to be evidence, they have not tested to see if evolution exists.
http://en.wikipedia.org/wiki/Scientific_theory
"Every genuine test of a theory is an attempt to falsify it, or to refute it. Testability is falsifiability; but there are degrees of testability: some theories are more testable, more exposed to refutation, than others; they take, as it were, greater risks."
The evidence is an attempt to prove the theory not an attempt to disprove, therefore not an acceptable test. If it could survive such a test, it would be a proper theory. But it can't survive a test b/c it can't be tested.
Exactly, explaining why you believe they were more fit does not refute that it is circular logic. The fact is you define that they are more fit based on the fact that they survive, and if they survive that means they are more fit.
Engimo said:Actually, I am. I'm a scientist - granted, not a biologst, but I keep myself apprised of the goings-on when it comes to Evolutionary theory.
Like I said, predictiveness of a scientific theory is not the sort of predictiveness that you think of usually when you hear the word. The predictive capability of a scientific theory doesn't necessarily have to apply to future events. For Evolution to be predictive, you have to be able to look at an environment under certain conditions and containing certain organisms with certain traits and be able to accurately say which ones are going to survive. Looking at the past, we can see what actually happened with different species and see if the history matches up with what Evolutionary Theory says will happen.
Yes, it can. The fossil record, mitochondrial DNA, and numerous other things provide evidence for Evolutionary Theory. That is the "testability" that is being referred to. There are a lot of scientific theories, especially in biology, that cannot be "tested" in the sense of going into a laboratory and performing an experiment, but we have evidence for them that supports it.
A "test" that would refute Evolutionary Theory would be finding a highly-evolved fossil in part of the fossil record that it should not be.
No, the logic doesn't go like that.
You're saying that the pattern of Evolutionary Theory goes like this:
1. Organisms survive because they are more fit.
2. They are more fit because they survive.
This is not the case. It is like this:
1. Organisms reproduce because they are more fit.
2. They are more fit because they have certain traits that allow them to survive longer in an environment.
See the different? It follows common sense, as well. If an organism is dead, it obviously cannot reproduce, so it will not pass down its genetic information. So, traits that cause an organism to die will be selected out because they will not be able to reproduce, while traits that allow organisms to survive better will be passed down more because they have more time to reproduce.
Still, this is research, not a test. What kind of scientific theories are tested in this manner?
And how do you decide that those traits are the reason they are more fit? Because they survived.
However, you are assuming that the organism is dead because it wasn't 'fit'. Therefore, that assumes that surviving organisms are alive because they were the fittest. Correct? So if you define the fittest by those who survive, of course it can't be proven wrong.
Engimo said:Outside of the realm of Physics and other highly testable fields, many of them. Evolution deals primarily with historical things, but it can also talk about what is going to happen. For example, look at the flu virus. The only reason that we need new flu shots every year is because of Evolution.
Sigh. No, the reason that the traits were beneficial is entirely contingent upon the environment and situation being discussed. Take my example of the flooded area that I mentioned before. If we know that an area flooded, we also know that those who cannot breathe underwater or live in flooded areas will not survive. That's all there is to natural selection. Go back and read my last post, there is no circular reasoning involved.
No, you're assuming the organism is dead because it died.
No more than there is a "debate" about whether the Earth is round. Oh, yes, the marginal, anti-science "just because I believe otherwise" crowd, be they flat-earthers or creationists/IDers are no more than that.HTColeman said:Well, there is much debate over whether evolution qualifies as a theory.
Both claims are untrue.It cannot be tested and it does not have "predictive qualities".
Well, take a look at this:Scientists cannot begin to say that under 'x' conditions 'y' will evolve into 'z' over 't' amount of years.
False. What is the source for this? You put it in quotes, so you got it from somewhere, but it wasn't in the link you provided (a creationist, un-scientific site), so where is this plagiarized from?Also it has circular reasoning;
"The concept of natural selection by survival of the fittest is the basic evolutionary mechanism. This concept does not qualify as a scientific principle, since fitness is equivalent to survival.
Sure it can. Your source has grave ignorance of the science that went into the formulation of the Scientific Theory of Evolution.Here we have a case of circular reasoning; no consistency or predictive value can be tested..
They survived because their evolutionary changes conferred a competitive advantage in carrying offspring to adulthood.According to this idea, organisms have survived through the evolutionary process because they are better fit, and the way one tells they are better fit is that they survive."
But none of that involves the validity of Evolution as a Scientific Theory like YOU claimed, so your claim remains a falsehood. You are still evidenced to bear FALSE WITNESS.HTColeman said:I take it that you are so involved in the scientific community that you are aware of the various debates within?
Notabout the validity of Evolution as a Scientific Theory, outside of the fringe ignoramuses.Besides I simply said much debate, I didn't say where, and there is much debate.
See example in the link of the previous post.So, how is it predictive? Furthermore, according to the definition from Wikipedia you posted, it is inherent to the definition of a theory.
Your claim is false.That is not a test, that is what they believe to be evidence, they have not tested to see if evolution exists.
Another false claim.http://en.wikipedia.org/wiki/Scientific_theory
"Every genuine test of a theory is an attempt to falsify it, or to refute it. Testability is falsifiability; but there are degrees of testability: some theories are more testable, more exposed to refutation, than others; they take, as it were, greater risks."
The evidence is an attempt to prove the theory not an attempt to disprove, therefore not an acceptable test.
A falsehood.If it could survive such a test, it would be a proper theory. But it can't survive a test b/c it can't be tested.
False.Exactly, explaining why you believe they were more fit does not refute that it is circular logic. The fact is you define that they are more fit based on the fact that they survive, and if they survive that means they are more fit.
Yes, see my previous link.HTColeman said:And can you provide evidence that puts certain conditions containing certain organisms and accurately saying which ones will survive?
All Scientific Theopries. Scientists look aty the data and determines that they then should see outcome X under certain conditions, and then research whether outcome X indeed occurs as predicted.Still, this is research, not a test. What kind of scientific theories are tested in this manner?
Nope.And how do you decide that those traits are the reason they are more fit? Because they survived.
Nope.However, you are assuming that the organism is dead because it wasn't 'fit'. Therefore, that assumes that surviving organisms are alive because they were the fittest. Correct?
But then, that is not the definition either.So if you define the fittest by those who survive, of course it can't be proven wrong.
steen said:No more than there is a "debate" about whether the Earth is round. Oh, yes, the marginal, anti-science "just because I believe otherwise" crowd, be they flat-earthers or creationists/IDers are no more than that.
Both claims are untrue.
Well, take a look at this:
http://www.gate.net/~rwms/EvoMutations.html
Particularly look at examples number 4 and 9.
Your claim is false.
False. What is the source for this? You put it in quotes, so you got it from somewhere, but it wasn't in the link you provided (a creationist, un-scientific site), so where is this plagiarized from?
Sure it can. Your source has grave ignorance of the science that went into the formulation of the Scientific Theory of Evolution.
They survived because their evolutionary changes conferred a competitive advantage in carrying offspring to adulthood.
But not the SAME flu (of course, viruses are not the best example of evolution, but lets skip that for the moment). Speciation is not needed for evolution to occur. I am not sure where you got the idea that evolution is all about new species, but perhaps that is the source for your apparent ignorance of the science?HTColeman said:The flu is only adapting to circumstances, it is still the flu, it has not evolved to something greater than the flu.
I don't base things on beliefs, but rather of factual evidence. And the evidence is for evolution, just like the evidence is for the earth as a globe. And the claims against either are equally ignorant and cooky-marginal.HTColeman said:So I guess you feel more secure in your belief by bashing those who believe otherwise.
The site won't work, could post examples 4 and 9?
4.) Adaptation to a Low Phosphate Chemostat Environment by a Clonal Line of Yeast
P.E. Hansche and J.C. Francis set up chemosats to allow evolution of a single clonal line of beer yeast in a phosphate limited (due to high pH) environment. (A chemostat is a device that allows the propagation of microorganisms in an extremely constant environment.) The yeast clones grew slowly for about the first 180 generations when there was an abrupt increase in population density. This was later shown to be due to better assimilation of the phosphate, presumably due to an improvement in the permease molecule. (Permease is an enzyme that controls what is allowed to come into the cell through the yeast's cell membrane.) After about 400 generations, a second improvement in cell growth rates occurred because of a mutation to the yeast's phosphatase (an enzyme that improves the cells ability to use phosphate). The phosphatase became more active overall, and its optimal pH (the pH where it is most active) was raised. Finally, a third mutant appeared after 800 generations that caused the yeast cells to clump. This raised the population density in the chemostat because individual cells were no longer being washed out of chemostat (which is one of the methods that the chemostat uses to maintain very uniform conditions) as quickly as they had prior to the mutation. (This is just speculation on my part, but I wonder if it wasn't under some similar conditions that multi-cellularity became favored over unicellularity - perhaps on a sea bed or river bottom.)9.) Changes in the substrate specificities of an enzyme during directed evolution of new functions.
This experiment was repeated, and the same mutations occurred, but in different orders. Also, in one replication, the processing of phosphate was improved by a duplication of the gene that produces phosphatase. This is experimental evidence of an extremely important mechanism in evolutionary history! It is also a particularly elegant experiment because not only was all of this adaptation shown to occur in clonal lines (descended from a single individual), but the authors also determined the exact mutations that caused the improved adaptations by sequencing the genes and proteins involved.
Francis, J.E., & Hansche, P.E. (1972) Directed evolution of metabolic pathways in microbial populations. I. Modification of the acid phosphatase pH optimum in Saccharaomyces cervisiae. Genetics, 70: 59-73.
Francis, J.E., & Hansche, P.E. (1973) Directed evolution of metabolic pathways in microbial populations. II. A repeatable adaptation in Saccharaomyces cervisiae. Genetics, 74:259-265. Hansche, P.E. (1975) Gene duplication as a mechanism of genetic adaptation in Saccharaomyces cervisiae. Genetics, 79: 661-674.
Hall BG
Biochemistry 1981 Jul 7 20:14 4042-9
Abstract
Wild-type ebg enzyme, the second beta-galactosidase of Escherichia coli K12, does not permit growth on lactose. As part of a study of the evolution of new enzymatic functions, I have selected, from a lacZ deletion strain, a variety of spontaneous mutants that grow on lactose and other beta-galactoside sugars. Single point mutations in the structural gene ebgA alter the enzyme so that it hydrolyzes lactose or lactulose effectively; two mutations in ebgA permit galactosylarabinose hydrolysis, while three mutations are required for lactobionic acid hydrolysis. Wild-type ebg enzyme and 16 functional mutant ebg enzymes were purified and analyzed kinetically to determine how the substrate specificities had changed during the directed evolution of these new functions. The specificities for the biologically selected substrates generally increased by at least an order of magnitude via increased Vmax and decreased Km for the substrate. These changes were very specific for the selected substrate, often being accompanied by decreased specificities for other related substrates. The single, double, or triple substitutions in the enzymes did not detectably alter the thermal stability of ebg enzyme.I searched on terms in the text and they didn't show up on the page you linked to. SO do you mind telling me where the text in quotes came from?Yeah, it was, so you obviously didn't look very well, and I guess by accusing me of plagiarizing, your proving your point?
By allowing them to be more successful at bringing offspring to adulthood.With hindsight, how do you determine that their traits gave them competitive advantage?
steen said:I don't base things on beliefs, but rather of factual evidence. And the evidence is for evolution, just like the evidence is for the earth as a globe. And the claims against either are equally ignorant and cooky-marginal.
The site won't work, could post examples 4 and 9?
4.) Adaptation to a Low Phosphate Chemostat Environment by a Clonal Line of Yeast
P.E. Hansche and J.C. Francis set up chemosats to allow evolution of a single clonal line of beer yeast in a phosphate limited (due to high pH) environment. (A chemostat is a device that allows the propagation of microorganisms in an extremely constant environment.) The yeast clones grew slowly for about the first 180 generations when there was an abrupt increase in population density. This was later shown to be due to better assimilation of the phosphate, presumably due to an improvement in the permease molecule. (Permease is an enzyme that controls what is allowed to come into the cell through the yeast's cell membrane.) After about 400 generations, a second improvement in cell growth rates occurred because of a mutation to the yeast's phosphatase (an enzyme that improves the cells ability to use phosphate). The phosphatase became more active overall, and its optimal pH (the pH where it is most active) was raised. Finally, a third mutant appeared after 800 generations that caused the yeast cells to clump. This raised the population density in the chemostat because individual cells were no longer being washed out of chemostat (which is one of the methods that the chemostat uses to maintain very uniform conditions) as quickly as they had prior to the mutation. (This is just speculation on my part, but I wonder if it wasn't under some similar conditions that multi-cellularity became favored over unicellularity - perhaps on a sea bed or river bottom.)9.) Changes in the substrate specificities of an enzyme during directed evolution of new functions.
This experiment was repeated, and the same mutations occurred, but in different orders. Also, in one replication, the processing of phosphate was improved by a duplication of the gene that produces phosphatase. This is experimental evidence of an extremely important mechanism in evolutionary history! It is also a particularly elegant experiment because not only was all of this adaptation shown to occur in clonal lines (descended from a single individual), but the authors also determined the exact mutations that caused the improved adaptations by sequencing the genes and proteins involved.
Francis, J.E., & Hansche, P.E. (1972) Directed evolution of metabolic pathways in microbial populations. I. Modification of the acid phosphatase pH optimum in Saccharaomyces cervisiae. Genetics, 70: 59-73.
Francis, J.E., & Hansche, P.E. (1973) Directed evolution of metabolic pathways in microbial populations. II. A repeatable adaptation in Saccharaomyces cervisiae. Genetics, 74:259-265. Hansche, P.E. (1975) Gene duplication as a mechanism of genetic adaptation in Saccharaomyces cervisiae. Genetics, 79: 661-674.
Hall BG
Biochemistry 1981 Jul 7 20:14 4042-9I searched on terms in the text and they didn't show up on the page you linked to. SO do you mind telling me where the text in quotes came from?
Abstract
Wild-type ebg enzyme, the second beta-galactosidase of Escherichia coli K12, does not permit growth on lactose. As part of a study of the evolution of new enzymatic functions, I have selected, from a lacZ deletion strain, a variety of spontaneous mutants that grow on lactose and other beta-galactoside sugars. Single point mutations in the structural gene ebgA alter the enzyme so that it hydrolyzes lactose or lactulose effectively; two mutations in ebgA permit galactosylarabinose hydrolysis, while three mutations are required for lactobionic acid hydrolysis. Wild-type ebg enzyme and 16 functional mutant ebg enzymes were purified and analyzed kinetically to determine how the substrate specificities had changed during the directed evolution of these new functions. The specificities for the biologically selected substrates generally increased by at least an order of magnitude via increased Vmax and decreased Km for the substrate. These changes were very specific for the selected substrate, often being accompanied by decreased specificities for other related substrates. The single, double, or triple substitutions in the enzymes did not detectably alter the thermal stability of ebg enzyme.
So you are trying to tell me, that you base the origin of all life on earth on some test on yeast (which the experiment is called an adaptation, not evolution) and enzymes? I looked at the website (it was my browser that had the problem) and most of the experiments described adaptations, not to mention the site is not a scientific site, just someones collection.
By allowing them to be more successful at bringing offspring to adulthood
So basically you prove they had competitive advantage by the fact their line survived?
steen said:I don't base things on beliefs, but rather of factual evidence. And the evidence is for evolution, just like the evidence is for the earth as a globe. And the claims against either are equally ignorant and cooky-marginal.
The site won't work, could post examples 4 and 9?
4.) Adaptation to a Low Phosphate Chemostat Environment by a Clonal Line of Yeast
P.E. Hansche and J.C. Francis set up chemosats to allow evolution of a single clonal line of beer yeast in a phosphate limited (due to high pH) environment. (A chemostat is a device that allows the propagation of microorganisms in an extremely constant environment.) The yeast clones grew slowly for about the first 180 generations when there was an abrupt increase in population density. This was later shown to be due to better assimilation of the phosphate, presumably due to an improvement in the permease molecule. (Permease is an enzyme that controls what is allowed to come into the cell through the yeast's cell membrane.) After about 400 generations, a second improvement in cell growth rates occurred because of a mutation to the yeast's phosphatase (an enzyme that improves the cells ability to use phosphate). The phosphatase became more active overall, and its optimal pH (the pH where it is most active) was raised. Finally, a third mutant appeared after 800 generations that caused the yeast cells to clump. This raised the population density in the chemostat because individual cells were no longer being washed out of chemostat (which is one of the methods that the chemostat uses to maintain very uniform conditions) as quickly as they had prior to the mutation. (This is just speculation on my part, but I wonder if it wasn't under some similar conditions that multi-cellularity became favored over unicellularity - perhaps on a sea bed or river bottom.)9.) Changes in the substrate specificities of an enzyme during directed evolution of new functions.
This experiment was repeated, and the same mutations occurred, but in different orders. Also, in one replication, the processing of phosphate was improved by a duplication of the gene that produces phosphatase. This is experimental evidence of an extremely important mechanism in evolutionary history! It is also a particularly elegant experiment because not only was all of this adaptation shown to occur in clonal lines (descended from a single individual), but the authors also determined the exact mutations that caused the improved adaptations by sequencing the genes and proteins involved.
Francis, J.E., & Hansche, P.E. (1972) Directed evolution of metabolic pathways in microbial populations. I. Modification of the acid phosphatase pH optimum in Saccharaomyces cervisiae. Genetics, 70: 59-73.
Francis, J.E., & Hansche, P.E. (1973) Directed evolution of metabolic pathways in microbial populations. II. A repeatable adaptation in Saccharaomyces cervisiae. Genetics, 74:259-265. Hansche, P.E. (1975) Gene duplication as a mechanism of genetic adaptation in Saccharaomyces cervisiae. Genetics, 79: 661-674.
Hall BG
Biochemistry 1981 Jul 7 20:14 4042-9I searched on terms in the text and they didn't show up on the page you linked to. SO do you mind telling me where the text in quotes came from?
Abstract
Wild-type ebg enzyme, the second beta-galactosidase of Escherichia coli K12, does not permit growth on lactose. As part of a study of the evolution of new enzymatic functions, I have selected, from a lacZ deletion strain, a variety of spontaneous mutants that grow on lactose and other beta-galactoside sugars. Single point mutations in the structural gene ebgA alter the enzyme so that it hydrolyzes lactose or lactulose effectively; two mutations in ebgA permit galactosylarabinose hydrolysis, while three mutations are required for lactobionic acid hydrolysis. Wild-type ebg enzyme and 16 functional mutant ebg enzymes were purified and analyzed kinetically to determine how the substrate specificities had changed during the directed evolution of these new functions. The specificities for the biologically selected substrates generally increased by at least an order of magnitude via increased Vmax and decreased Km for the substrate. These changes were very specific for the selected substrate, often being accompanied by decreased specificities for other related substrates. The single, double, or triple substitutions in the enzymes did not detectably alter the thermal stability of ebg enzyme.
So you are trying to tell me, that you base the origin of all life on earth on some test on yeast (which the experiment is called an adaptation, not evolution) and enzymes? I looked at the website (it was my browser that had the problem) and most of the experiments described adaptations, not to mention the site is not a scientific site, just someones collection.
By allowing them to be more successful at bringing offspring to adulthood
So basically you prove they had competitive advantage by the fact their line survived?
Oh yeah, FYI, I won't respond to 'False', 'Not True', etc. Those aren't arguments, just a waste of space.
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